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analysis of management and outcome in 340 consecu-tive cases. Recent datasuggest that the vulnerability of CA1 neurons to meta-bolic stress plays a pivotal part in the pathophysiologiccascade, leading to an impairment of hippocampal func-tion during TGA (Bartsch and Deuschl, 2010)

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PKC-theta is a negative regulator of Tregsuppressive function by virtue of its role during responding T cell synapse formation andsubsequent T cell activation, which is the result of PKC-theta activation of the NF-kBpathway.

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